Gordon Johnson, Extension Vegetable & Fruit Specialist; email@example.com
Blossom end rot (BER) is showing up again this year in peppers and tomatoes. BER is a disorder where developing fruits do not have enough calcium for cell walls, cells do not form properly, and the fruit tissue at the blossom end collapses, turning dark in color. Calcium moves through cation exchange with water movement in the fruit, so the end of the fruit will be the last to accumulate calcium. Larger fruits and longer fruits are most susceptible. With fruits, the rapid cell division phase occurs early in the development of the fruit and if calcium accumulation in the fruit is inadequate during this period, BER may occur. While it may not be noticed until the fruit expands, the deficiency has already occurred and cells have already been negatively affected. We most commonly see signs of blossom end rot on fruits many days after the calcium deficiency has occurred.
Understanding blossom end rot also requires an understanding of how calcium moves from the soil into and through the plant. Calcium moves from the soil exchange sites into soil water and to plant roots by diffusion and mass flow. At plant roots, the calcium moves into the xylem (water conducting vessels), mostly from the area right behind root tips. In the xylem, calcium moves with the transpirational flow, the movement of water from roots, up the xylem, and out the leave through stomata. Calcium is taken up by the plant as a divalent cation, which means it has a charge of +2. It is attracted to negatively charged areas on the wall of the xylem, and for calcium to move, it must be exchanged off the xylem wall by other positively charged cations such as magnesium (Mg++), potassium (K+), ammonium (NH4+), or additional calcium cations (Ca++). This cation exchange of calcium in the xylem requires continuous movement of water into and up through the plant. It also requires a continuous supply of calcium from the soil.
In general, most soils have sufficient calcium to support proper plant growth. While proper liming will insure there is adequate calcium, it is not the lack of calcium in the soil that causes blossom end rot in most cases. It is the inadequate movement of calcium into plants that is the common culprit. Anything that impacts root activity or effectiveness will limit calcium uptake. This would include dry soils, saturated soils (low oxygen limits root function), compaction, root pathogens, or root insect damage. In hot weather on black plastic mulch, roots can also be affected by high bed temperatures. Low pH can also be a contributing factor. Calcium availability decreases as pH drops, and below a pH of 5.2 free aluminum is released, directly interfering with calcium uptake. Again, proper liming will insure that this does not occur. Applying additional calcium as a soil amendment, above what is needed by normal liming, will not reduce blossom end rot.
In the plant, there is a “competition” for calcium by various plant parts that require calcium such as newly forming leaves and newly forming fruits. Those areas that transpire the most will receive more calcium. In general, fruits have much lower transpiration than leaves. In hot weather, transpiration increases through the leaves and fruits receive lower amounts of calcium. High humidity will reduce calcium movement into the fruit even more. Tissue tests will often show adequate levels of calcium in leaf samples; however, fruits may not be receiving adequate calcium. In addition, in hot weather, there is an increased risk of interruptions in water uptake, evidenced by plant wilting, when transpirational demand exceeds water uptake. When plants wilt, calcium uptake will be severely restricted. Therefore, excess heat and interruptions in the supply of water (inadequate irrigation and/or rainfall) will have a large impact on the potential for blossom end rot to occur. Proper irrigation is therefore critical to manage blossom end rot.
As a positive cation, there is “competition” for uptake of calcium with other positive cations. Therefore, if potassium, ammonium, or magnesium levels are too high in relation to calcium, they can reduce calcium uptake. To manage this, do not over-fertilize with potassium or magnesium and replace ammonium or urea sources of nitrogen with nitrate sources.
Applying additional soluble calcium through irrigation, especially drip systems, can reduce blossom end rot to some degree if applied prior to and through heat events and if irrigation is applied evenly in adequate amounts. Foliar applications are much less effective because fruits do not absorb much calcium, especially once a waxy layer has developed, and calcium will not move from leaves into the fruit (there is little or no phloem transport).
In conclusion, the keys to controlling blossom end rot are making sure roots are actively growing and root systems are not compromised, soil pH is in the proper range, and irrigation is supplied in an even manner so that calcium uptake is not interrupted. Supplemental calcium fertilization will only marginally reduce blossom end rot if water is not managed properly.
Another calcium disorder that is found in peppers is called “stip”. These spots on peppers occur later in the year, commonly in the late summer or fall, during cool, humid conditions. Under these conditions, calcium movement into the fruit is uneven, leading to localized collapse of cells, causing the spotting. Again, making sure adequate calcium is moving in the plant is critical to control stip.